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Although insulin resistance (IR) plays a central role in metabolic diseases, in the liver, insulin- or substrate-driven de novo lipogenesis (DNL) promotes triglyceride accumulation through multiple complex regulatory mechanisms, including specific transcription factors, regardless of whether IR is primary or not

Its origins in gastric juice hint at one of its most powerful roles: gut protection and healing

Glutathione acts as a protective shield, taking the damage and saving the surrounding cell components from oxidation

We also explored the potential therapeutic opportunities for targeted metabolism to rescue the phenotype of VSMCs