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It already activates GLP-1 and GIP (plus glucagon), so adding another agonist is redundant and raises risk

glutathione peroxidase, GPx) and non-enzymatic scavengers (e.g., glutathione, vitamin E) (Lushchak, 2014)

This article explores the potential of GLP-1 agonist drugs in treating Alzheimers disease (AD) by targeting dysfunctions in all major brain cell types