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Despite initial enthusiasm for cuproptosis as a novel pathogenetic mechanism [27,51,54] , the molecular and metabolic complexities underlying copper-dependent hepatocellular injury in Wilson disease remain unresolved, especially since the role of ROS and reactive copper intermediates was not originally included in the cuproptosis framework [51,54]

In the stomach, hydrochloric acid and pepsin release B12 from food proteins, after which intrinsic factora glycoprotein produced by gastric parietal cellsbinds to the vitamin

These peptides encourage the release of growth hormone, but they do so either by mimicking or stimulating the release of another peptide called ghrelin