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Mutations in the Casitas B-cell lymphoma (Cbl)-binding domain are demonstrated to be oncogenic because binding of Cbl to Y1003 causes Met ubiquitination that is vital to maintenance of physiological Met activation and prevention of continued activation of Met (Peschard and Park, 2007)
Keep it separate so side effects and dose response are easy to track
The rationale for stacking GHK-Cu vs NAD+ together is additive rather than synergistic: GHK-Cu handles structural repair while NAD+ handles bioenergetics
It's the community's shorthand for the idea that BPC-157 and TB-500 together create a more complete healing response than anything else available
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