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It is a well-documented cause of CL dysfunction, which is oxidative stress, or the excessive generation of reactive oxygen species (ROS) (228)

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This HO-1-dependent cytoprotective mechanism may explain much of BPC-157s observed ability to protect cells against ischaemia-reperfusion injury and oxidative damage across multiple tissue types simultaneously

Effect of oxidized B3-crystallin peptide (152-166) on thermal aggregation of bovine lens -crystallins: identification of peptide interacting sites