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[110] found that methyl 3,5-dicaffeoylquinine increased TYR activity and melanin production in B16F10 through a master regulator of melanocyte activity, MITF, and methyl 3,5-dicaffeoylquinine-mediated MITF activation relies on cAMP/PKA signalling and p38 MAPK phosphorylation

However, pathological levels of ROS detected in semen of infertile men are more likely a result of increased ROS production, rather than a reduced antioxidant capacity of the seminal plasma (Zini et al., 1993)

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The compound-level mechanisms are covered in KLOW Peptide Mechanisms