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Interestingly, examination of the peptide sequence revealed that P5 N-terminal residues are distinct from residues 18 of the endogenous hormone or Ex4 suggesting that extensive sequence modifications rather than minimal structural changes (single amino acid change) are required to produce biased agonism at the GLP-1R
ER stress sensor XBP1 controls anti-tumor immunity by disrupting dendritic cell homeostasis
There is currently no approved human therapeutic use
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