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The linear decline was recorded for 10 s, with the initial absorbance being set to 0

Pro-inflammatory cytokines activate chondrocytes to release catabolic factors, which impede the production of COL2, proteoglycans, and other synthetic metabolic components, hence facilitating chondrocyte catabolic processes and the degradation of articular cartilage [39,40]

Additionally, TB-500 presentation following alkali injury may reduce the expression of chemoattractants for polymorphonuclear neutrophils (PMNs), MIP-2, and KC in mouse corneas, possibly resulting in reduced PMN infiltration