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Despite all the beneficial effects of IGF-I described up to now and the following ones reporting its role in aging-related pathologies as musculoskeletal, neurodegenerative, cardiovascular diseases, IGF-I has been paradoxically negatively related to lifespan[266, 267], probably due, at least in part, to its downstream molecule Akt and its interactions with FOXO, mTOR and SIRT-1, among others[268, 269]
in Microbiology, Immunology, and Molecular Genetics with a Minor in Biomedical Research from the University of California, Los Angeles
Esta alteracin se debe principalmente a tres factores: Estrs oxidativo crnico , que agota las reservas de GSH al neutralizar un exceso de radicales libres
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