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On the basis of the ability of GLP-1RA/tesaglitazar to induce neuronal activation in the hypothalamus, and recent data indicating that PPAR might affect systemic energy metabolism via hypothalamic neurocircuitries 5,6 , we next assessed the response of the hypothalamic proteome of DIO mice 10 h after single peripheral (s.c.) drug treatment using liquid chromatographymass spectrometry (LCMS)

In the eWAT, no group separation was observed in the PCA, and only five and three proteins were upregulated by GLP-1GIPLani and GLP-1GIP, respectively (Extended Data Fig

This response leads to an increase in molecular chaperones, enhancing protein folding efficiency or stabilizing protein conformations, thereby reducing proteotoxicity

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