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Endoplasmic reticulum chaperones GRP78 and calreticulin prevent oxidative stress, Ca2+ disturbances, and cell death in renal epithelial cells
Injections skip that process entirely and go straight to work
In children with autism, neural progenitor cells show accumulation of iron and high levels of oxidative stress, as well as increased risk of ferroptosis, which is iron-dependent programmed cell death characterized by lipid peroxidation
The many roads to mitochondrial dysfunction in neuroimmune and neuropsychiatric disorders
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